Are there any alternatives to niacin?

10. September 2006 William Davis (0)
In the Track Your Plaque program, we tend to rely a great deal on niacin. When used properly, 90-95% of people will do just fine and achieve their lipid and lipoprotein goals with the help of niacin, along with their other efforts.

Unfortunately, around 5% of people simply can't take niacin without intolerable "hot flush" effects, or occasionally excessive skin sensitivity--itching, burning, etc.

Why does this happen? These 5% tend to be "rapid metabolizers" of niacin, i.e. they convert niacin (nicotinic acid, or vitamin B3) into a metabolite called nicotinuric acid. Nicotinuric acid is the compound responsible for the skin flush. Most people can slow or reduce the effects of nicotinuric acid by:

--Taking niacin with dinner, so that food slow tablet dissolution.

--Taking with plenty of water. Two 8-12 oz glasses usually eliminates the flush entirely in most people.

--Taking with an uncoated 325 mg tablet of aspirin in the first few weeks or months. Eventually, you will need to revert back to a better stomach tolerated dose of 81 mg, preferably enteric coated. But a full 325 mg uncoated can really help in the beginning, or when you have any niacin dose increases, e.g., 500 mg to 1000 mg.

But even with these very effective strategies, some people still struggle. That's when the question arises: Are there any alternatives to niacin?

Well, it depends on why niacin is being used. If you and your doctor are using niacin for:

Raising HDL--Then weight loss to your ideal weight; reduction of processed carbohydrates, especially wheat products; avoidance of hydrogenated ("trans") fats; a glass or two of red wine per day; dark chocolates (make sure first ingredient is chocolate or cocoa, not sugar), 40 gm per day; fish oil; exercise; other prescription agents (fibrates like Tricor; TZD agents for diabetes; cilostazol (Pletal)). Niacin is by far the most effective agent of all, but, if you're intolerant, raising HDL is still possible through a multi-faceted effort.

Reduction of small LDL--The list of effective strategies is the same as for raising HDL, but add raw almonds (1/4-1/2 cup per day), oat bran and other beta-glucan rich foods like oatmeal. Reduction of processed carbohydrates is especially important to reduce small LDL.

Reduction of Lipoprotein(a)--This is a tricky one. For men, testosterone and DHEA are effective alternatives; for women, estrogen and perhaps DHEA. Hormonal preparations of testosterone and estrogen are stricly prescription; DHEA is OTC. I have not seen the outsized benefits on lipoprotein(a) claimed by Rath et al by using high-dose vitamin C, lysine, and profile, unfortunately. We are clearly in need of better alternatives to treat this difficult and high-risk disorder.

Reduction of triglycerides/VLDL/IDL--I lump these three together since they all respond together. If you're niacin intolerant, maximixing your fish oil can be crucial for reduction of these patterns using doses above the usual starting 4000 mg per day (providing 1200 mg EPA+DHA). Reduction of processed carbohydrates, eimination of processed foods that contain high-fructose corn syrup, and weight loss to ideal weight are also very effective. "Soft" strategies with modest effects include green tea (>6 cups per day) or theaflavin 600-900 mg/day; raw nuts like almonds, walnuts, and pecans; exercise; soy protein.

Reduction of LDL--Lots of alternatives here including oat bran (3 tbsp per day), ground flaxseed (3 tbsp per day), soy protein (25 grams per day), Benecol butter substitute (for stanol esters), soluble fibers like pectin, psyllium, glucomannan; raw nuts like almonds, walnuts, and pecans.

In future, should torcetrapib become available (by prescription), this will add to our available tools for these areas when niacin can't be used. Until now, the alternatives to niacin depend on what you and your doctor are trying to achieve. In the vast majority of cases, HDL, small LDL, triglyceride, etc. goals for heart scan score control can be achieved, even when niacin is not well tolerated.
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If you need a reason to quit smoking...

If you need a reason to quit smoking...

26. April 2006 William Davis (0)
If you've read Track Your Plaque, you already know my feelings about smoking and coronary plaque. Smoke, and you will lose the battle for control over coronary plaque growth--it will grow and grow until catastrophe strikes.

Nonetheless, this is not sufficiently motivating for some people.

If you need more motivation to quit smoking, just take a look at your heart scan sometime, accompanied by either one of the doctors or technicians at the scan center you choose. After you've had an opportunity to look at your coronary arteries, take a look at the lungs. The heart is in the middle and the lungs are the two large black areas on either side of the heart. (They're not really black; that's just the way the images are color-coded.)

Smokers will see large cavities in their lungs--literally, half-inch to one-inch wide holes that contain only air. Many of them. These represent remnants of lung tissue, digested away and now useless from the damage incurred through smoking.

Non-smokers should see uniform lung tissue without such cavities.

What surprised me early on in my heart scan experience was how little smoking exposure was required to generate these cavities. A 40-year old, for instance, who smoked a half-pack per day for 10 years would have them. Heavier smokers, of course, showed far more extensive cavities.

Officially, these cavities are called "emphysematous blebs", meaning the scars of the lung disease, emphysema.

When I've pointed out these cavities or emphysematous blebs to patients, 9 out of 10 times they immediately become non-smokers. Commonly, they'd exclaim, "I had no idea I was really damaging my lungs!" Most admitted that they were awaiting some bona fide evidence that they were truly doing some harm to their bodies. Well, that's it.

Give it a try if you're struggling.
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Yet another reason to avoid fructose

Yet another reason to avoid fructose

6. December 2008 William Davis (5)
Have you seen the Corn Refiners Association commercial campaigns to educate the American public on the safety of fructose? If you haven't, you can view these interesting specimens on You Tube:

"Get the facts--You're in for a sweet surprise: Fructose is safe in moderation!"

Two Moms

Two lovers


Beyond the fact that fructose stimulates liver production of glycerol, which thereby increases liver VLDL production and raises blood levels of triglycerides; likely stimulates appetite; increases cholesterol levels; fructose has also been clearly implicated in increasing blood levels of uric acid.

Uric acid is the substance that, in some people, precipitates in joints and causes gout, the painful inflammatory arthritis that has been increasing in prevalence over the last four decades since the introduction of fructose in 1967. While blood levels of uric acid in the early part of the 20th century averaged 3.5 mg/dl, more recent population assessments have averaged 6.0 mg/dl or higher. (Non-human mammals who don't eat processed foods, drink fruit drinks or beer, and don't eat candy have uric acid levels of <2.0 mg/dl.)

Uric acid is looking like it may prove to be an important risk factor for coronary disease and atherosclerotic plaque. It is no news that people with higher blood levels of uric acid are more likely to experience adverse cardiovascular events like heart attack. People with features of the metabolic syndrome also have higher uric acid blood levels; the more characteristics they have, the higher the uric acid level. However, the prevailing view has been that uric acid is simply an accompaniment of these processes, but not causal.

However, more recent observations suggest that increased levels of uric acid may instead be a cause of metabolic syndrome and high blood pressure.

Increased blood levels of uric acid have been shown to:

--Increase blood pressure
--Induce kidney damage (even in the absence of uric acid kidney stones)
--Antagonize insulin responses

A diagnosis of gout is not required to experience all of the adverse phenomena associated with uric acid. (For not entirely clear reasons, some people, perhaps based on pH or other factors, are more prone to trigger crystallization of uric acid in joints, similar to the phenomena of sugar crystallization when making rock candy.)

Which brings us back to fructose, a sweetener that clearly substantially increases uric acid levels. I suppose that the mothers and lovers in the Corn Refiners' commercials are right to a degree: Our kids will survive, as will you and I, despite increases in triglycerides, enhanced diabetic tendencies, amplified appetites, and increased uric acid due to fructose in our diet. We will also likely survive despite being 100 lbs overweight, partly due to the effects of fructose.

But if long-term health is your desire for you and your family, fructose has no role whatsoever to play.

Interestingly, the obviously expensive and slick ad campaigns from the Corn Refiners' videos have triggered some helpful video counterarguments:

High-fructose corn syrup
Conspiracy for Fat America
High-fructose corn syrup truth


A full discussion of uric acid, the scientific data behind uric acid as a coronary risk factor, and the nutritional means to reduce uric acid will be the topic of a thorough discussion in an upcoming Special Report on the Track Your Plaque website.
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Comments (5) -

  • Kevin

    12/12/2008 8:02:00 PM |

    There was an article this week in USA Today about new research pertaining to high fructose corn syrup (independent of your uric acid argument). Check it out here: http://www.usatoday.com/news/health/2008-12-08-fructose-corn-syrup_N.htm

    -Kevin on behalf of the Corn Refiners Association

  • BenignaMarko

    12/18/2008 5:28:00 PM |

    This all sounds doable.  It does seem that fructose is causing such health problems that the FDA should make sure it is removed from products.  We all know that we consume too much everything, so, can't they make the stuff without the fructose?  There is so much to worry about in recent years, we might as well not eat.
    Benigna Marko

  • Anonymous

    1/4/2009 10:36:00 PM |

    I don't deny the research posted in this post.  However, things are not as simple as they seem.

    I have done quite a lot of research on Multiple Sclerosis, and high levels of uric acid are actually implicated in LOWER levels of MS.  Uric acid actually can work as an antioxidant in the body; gout and MS are almost mutually exclusive.  You will almost never see someone who has gout having MS, and vice versa.

    In fact, increasing uric acid in MS patients has been shown to DECREASE RELAPSE RATES (see abstract below.)

    So, things are not as simple as they appear.  

    Just thought I'd post this information.

    -gb

    FROM MEDLINE


    5: Vojnosanit Pregl. 2006 Oct;63(10):879-82.Links
        Therapeutic value of serum uric acid levels increasing in the treatment of multiple sclerosis.
        Toncev G.

        Clinical Center Kragujevac, Center of Neurology, Kragujevac, Srbija.

        BACKGROUND/AIM: Uric acid was successfully used in both, prevention and treatment of the animal model of multiple sclerosis (MS). Recently it has been shown that inosine, a ribosylated precursor of uric acid, might be used to elevate serum uric acid levels in MS patients. The aim of this study was to evaluate the safety and efficacy of oral inosine as a single drug treatment in patients with MS. METHOD: We administered inosine orally to 32 MS patients from 2001-2004 year at doses from 1-2 g daily (given twice) depending on the pretreatment serum uric acid levels. The mean follow-up interval was 37.69+/-6.55 months. The other 32 MS patients, without any treatment except for a relapse period (matched by age, sex, duration of disease and functional disability), were used as controls. The follow-up interval of these patients was 36.39 +/- 2.68 months. The neurological disability was evaluated by the Expanded Disability Status Scale score (EDSS). RESULTS: During the observed period the treated MS patients were found to have the lower relapses rate than the non-treated MS patients (Chi-square test, p = 0.001). None of the patients have showed any adverse effect of inosine treatment. The non-treated MS patients were found to have a higher increasing in the mean EDSS score than the treated ones (two-way ANOVA-repeated measures/factor times, p = 0.025). CONCLUSION: Our results suggested that the treatment approaches based on the elevation of serum uric acid levels might prove beneficial for some MS patients


    1: Eur J Neurol. 2008 Apr;15(4):394-7. Epub 2008 Feb 26.Click here to read Links
        Variation of serum uric acid levels in multiple sclerosis during relapses and immunomodulatory treatment.
        Guerrero AL, Martín-Polo J, Laherrán E, Gutiérrez F, Iglesias F, Tejero MA, Rodríguez-Gallego M, Alcázar C.

        Neurology Unit, Hospital Río Carrión, Palencia, Spain. aguerrero@hcuv.sacyl.es

        Uric acid (UA), a product of purine metabolism, may be an antioxidant, perhaps acting as a scavenger of peroxynitrite. Patients with gout have a reduced incidence of multiple sclerosis (MS). A number of studies found that patients with MS have low serum levels of UA, although it has not been established whether this represents a primary deficit or a secondary effect. UA has also been proposed as a marker of disease activity and response to immunosuppressive or immunomodulatory treatment. We retrospectively reviewed 83 relapsing-remitting or secondary progressive MS patients (64 females and 19 males) followed in our Neurology Unit. We collected data concerning demographic variables as age and sex, and clinical variables as age of onset, clinical type, disease duration, EDSS score and total number of relapses. We considered UA levels in three different situations: during a relapse, during remission period and during remission period under immunomodulatory treatment [Interferon Beta 1a i.m. (Avonex; Biogen Idec Inc., Cambridge, MA, USA), Interferon Beta 1a s.c. (Rebif; Serono Europe Limited, London, UK), Interferon Beta 1b (Betaferon; Bayer Schering Pharma AG, Berlin, Germany) or Glatiramer Acetate (Copaxone; TEVA Neuroscience LLC, Kansas City, MO, USA)]. A Wilcoxon matched pairs test was carried out to determine differences between groups. A P-value less than 0.05 was considered statistically significant. In 33 patients, we were able to compare at least one UA value obtained during a relapse with at least one when remission without treatment. Mean serum UA levels were significantly lower when measured during a relapse (r: 0.39, P: 0.024). In 27 cases, we compared at least one remission value without treatment with at least one obtained during remission and immunomodulatory treatment. Mean serum UA levels significantly increased when determined during Interferon Beta or Glatiramer Acetate therapy (r: 0.84, P < 0.001). Although we do not know exactly whether and how UA is involved in MS pathogenesis, our data suggest that UA might reflect disease activity or treatment response in MS.

  • karl

    2/28/2009 3:56:00 AM |

    I read that Fructose is 10 times more reactive than glucose in forming AGE - Advanced Glycogen End-products - the process thought to start CAD.

    I avoid all sugar - but wonder if the extra 5-10% might make a difference.

  • Free Teleconferencing

    2/22/2011 11:49:58 AM |

    Very... Nicee... Blog.. I really appreciate it... Thanks..Smile

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To track small LDL, track blood sugar

To track small LDL, track blood sugar

24. December 2009 William Davis (28)
Here's a trick I learned after years of fussing over people's small LDL.

To gain better control over small LDL, follow blood sugars (blood glucose).

When you think about it, all the foods that trigger increases in blood sugar also trigger small LDL. Carbohydrates, in general, are the most potent triggers of small LDL. The most offensive among the carbohydrates: foods made with wheat. After wheat, there's foods made with cornstarch, sucrose (table sugar), and the broad categories of "other" carbohydrates, such as oats, barley, quinoa, sorghum, bulghur, etc.

Assessing small LDL requires a full lipoprotein assessment in which small LDL particles are measured (NMR, VAP, GGE). Not the easiest thing to do in the comfort of your kitchen.

However, you can easily and now cheaply check your blood sugar. Because blood sugar parallels small LDL, checking blood sugar can provide insight into how you respond to various foods and know whether glucose/small LDL have been triggered.

Here's how I suggest patients to do it:

1) Purchase an inexpensive blood glucose monitor at a discounter like Walmart or Walgreen's. You can buy them now for about $10. They're even sometimes free with promotional offers. You will also need to purchase lancets and test strips.

2) With a meal in question, check a blood sugar just prior to the meal, then again 60 minutes after finishing the meal. Say, for example, your pre-meal blood sugar is 102 mg/dl. You eat your meal, check it 60 minutes after finishing. Ideally, the postprandial (after-meal) blood sugar is no more than 102 mg/dl, i.e., no higher than pre-meal.

Perhaps you're skeptical that oatmeal in skim milk with walnuts and raisins will do any damage. So you perform this routine with your breakfast. Blood sugar beforehand: 100 mg/dl. Blood sugar 1 hour post: 163 mg/dl--Uh oh, not good for you. And small LDL will be triggered.

This approach is not perfect. It will not, for example, identify "stealth" triggers of blood sugar and small LDL like pasta, for the same reasons that pasta has a misleadingly low glycemic index: sugars are released slowly and not fully evident with the one-hour blood sugar.

Nonetheless, for most foods and meals, tracking your one-hour postprandial blood sugar can provide important insight into your individual susceptibility to sugar and small LDL-triggering effects.
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Comments (28) -

  • Anonymous

    12/23/2009 8:05:52 PM |

    Would glycated hemoglobin also be an accurate way to track small LDL? Just thinking it may be easier to get that tested, which should give a decent account of sugar intake for the past several months, than measure glucose daily. Although I'm not sure if it correlates to small LDL as well.

    But if so, what is an optimal glycated hemoglobin for non-diabetic types?

  • TeDWooD

    12/23/2009 10:59:14 PM |

    I liked what you put about the blood sugar being released slowly in meals like pasta. When you take you blood pressure test you should be very relaxed as well.
    I have wrote about blood pressure highs and lows in my blog, and what foods are recommended. You can find it here:
    http://thesuccessfulmale.blogspot.com/2009/11/stop-blood-pressure-highs-and-lows.html

  • Jim Purdy

    12/24/2009 1:26:58 AM |

    I have another tracking device which I like much better than my blood glucose meter

    A finger-tip pulse oximeter!

    I sometimes tend to have tachycardia and very unpleasant chest pains, and I have often used my finger-tip pulse oximeter to see what's going on.

    Until a few months ago, I would often have chest pains, emergency room visits, and one 9-1-1 ambulance call because I thought I was having heart attacks.

    All those ER visits got expensive, especially when the hospitals insisted on keeping me for 2-3 days each time to run zillions of dollars of tests.

    Often those attacks followed a greasy meal with something like bacon double cheeseburgers, and I "knew" that the fat was the culprit.

    However, after several low-carb bloggers directed my attention to the buns instead of the meat, I used my pulse oximeter to identify the problem foods.

    Yes, indeed, it was carbs, especially things like bread and -- much to my surprise -- ordinary breakfast cereals. I still miss my sandwiches and my Corn Chex, but I don't miss all those emergency room visits.

    I love my finger-tip pulse oximeter!

  • DrStrange

    12/24/2009 2:05:57 AM |

    Important to note that blood sugar monitors for home use are VERY approximate and are only accurate by plus/minus 20%.  Also, may vary widely between two readings taken seconds apart. I usually take 3 readings, bang bang bang, toss out any far outlier and average the what's left.  Still approximate but it makes me feel like it is more accurate ;)

  • Anonymous

    12/24/2009 2:20:43 AM |

    Wow, and this is news:

    http://news.bbc.co.uk/1/hi/health/8426591.stm

    -just in

    Another 'bad' cholesterol linked to heart disease found

  • Anne

    12/24/2009 8:15:49 AM |

    Dear Dr Davis,

    You have been writing about Lp(a) for years....why is it that the so called 'mainstream' medical profession appears only to have discovered it very recently ? Here in today's BBC news: 'Another 'bad' cholesterol linked to heart disease found':  http://news.bbc.co.uk/1/hi/health/8426591.stm

    Happy Christmas !
    Anne

  • vin

    12/24/2009 9:54:25 AM |

    Dr. Davis.
    If I am not mistaken I remember you saying that oats contributed to forming large LDL particles and not the small LDL.

    I am unable to find the article. Maybe you can reproduce that article.

  • Kurt

    12/24/2009 1:05:14 PM |

    I did this per your previous suggestion. I took one-hour and two-hour postprandial readings. My blood sugar never rose above 122, and was back down near fasting level after two hours. My diet contains a moderate amount of whole grains but almost no sugar and no refined grains.

    You could clearly see the difference between a meal of, say, salmon and vegetables, which barely raised my blood sugar above fasting, and one of chicken and brown rice, which would raise it to 110 - 120. It was a useful tool for assessing my diet.

  • Chloe

    12/24/2009 7:02:48 PM |

    Good information.  I have no health insurance and make too much money to qualify for public assistance, yet do not make enough to pay for many (close to all) tests.  I do participate in GrassRootsHealth for vitamin D (level from 7 to now 94), so I use anything at home I can to monitor what I can.

    The ReliOn Micro glucometer from Wal-Mart is a great little meter. It runs $12 for the kit which includes a few lancets and a lancing device.  The strips are the "sippy" kind and require the smallest sample on the market, just a dot of blood, and they are the least expensive strips on the market at $21.75 for 50 count.  I use them occasionally to track my own BG levels.

    Reason I purchased the meter?  The story is familiar:  Too many grain carbs recommended as the "good diet" which led to obesity and then diabetes.  That was my story, too, but the meter was purchased for my cat, Kipper.  The vets are in on this BS too.  All that expensive Eukanuba, Science Diet, vet prescribed grain diet and now my baby is a diabetic.  I regret listening to that for years.  

    I prick his ear twice a day, give him 1U of Levemir insulin, and then I check myself, occasionally.  Grains very, very bad.  Protein and fat very, very good.  Kipper's and my blood glucose levels are normal now, but he still needs insulin.  I am currently diet controlled.  Glad to know my diet control yields heart-healthy effects, too.

    Recently I had a yen for cherry tomatoes and overindulged.  We are talking about a cup more than usual.  Checked by meter this produced a 15 point increase over normal.  

    That cheap but reliable meter from Wal-Mart, if used (sometimes we turn off the internal prompt to check because we do not want to check) is a great tool and for me now a multipurpose tool.

  • Anonymous

    12/24/2009 7:29:10 PM |

    I'm concerned regarding this statement:

    "And the broad categories of "other" carbohydrates, such as oats, barley, quinoa, sorghum, bulghur, etc."

    In your Cheerios post

    http://heartscanblog.blogspot.com/2008/04/cheerios-and-heart-health.html

    you have actually touted oats by showing a study that pit it against wheat and it was shown to actually reduce LDL; this makes sense considering oats are a source of soluble fiber.

    "High-fiber oat cereal compared with wheat cereal consumption favorably alters LDL-cholesterol subclass and particle numbers in middle-aged and older men"

    http://www.ajcn.org/cgi/content/full/76/2/351

    Heres also a study that showed improved blood glucose response at breakfast that followed an evening in which barley was consumed:

    http://www.nature.com/ejcn/journal/v60/n9/abs/1602423a.html

  • Anonymous

    12/24/2009 9:28:39 PM |

    Noting that your readership is growing internationally, it might be useful for people to understand that the units of blood test measurement that are used quite frequently here in this blog are different in the US from most other places worldwide. These conversion factors may prove useful:

    Cholesterol (total,LDL,HDL): 38.7 (eg HDL of 60 mg/dl (US) is 1.55 mmol/L elsewhere)
    Triglycerides: 88.6 (eg reading of 60 mg/dl (US) is 0.68 mmol/L elsewhere)
    Blood Sugar: 18 (eg FBS of 90 mg/dl (US) is 5 mmol/L elsewhere)

  • Dr. William Davis

    12/24/2009 11:36:27 PM |

    Anon--

    The drawback of HbA1c is that the feedback is not immediate. You cannot use it to gain feedback on a particular food or behavior.

  • Dr. William Davis

    12/24/2009 11:38:04 PM |

    Chloe--

    I, too, learned this lesson with my pets. I have two Boston terriers who gained weight little by little on the cornstarch-first ingredient dog food I was feeding them. Now, choosing dog foods that are principally meat has finally allowed them to control their weight.

    I'm impressed with your cat's blood sugar checking!

  • Dr. William Davis

    12/24/2009 11:38:29 PM |

    Thanks for the conversion factors, Anon.

  • Vladimir

    12/25/2009 12:45:33 AM |

    Dr. Davis, I'm wondering if it's the total blood sugar that correlates with LDL, or the rise in blood glucose.  So, for example, if one has a fasting level of 95 and rises to 120 after a meal, is that better/worse than starting at 75 and rising to 105 -- in terms of increase in LDL?

  • Anonymous

    12/25/2009 7:11:36 PM |

    Jim Purdy,
    Can you give some details on how you use your fingertip pulse oximeter to identify problem foods?  What kind of readings do you see and when?  How do the readings correlate with high or low blood sugar?

    Thanks!

  • Peter

    12/26/2009 1:58:05 PM |

    Chloe,
       A friend of mine started giving his diabetic cat low carb catfood and the cat, now fine, went into insulin shock because her blood sugar had returned to normal unbenounced to my friend. Now, she is fine on no more insulin shots.

  • Jim Purdy

    12/26/2009 3:19:53 PM |

    Anonymous asked me:
    "Jim Purdy, Can you give some details on how you use your fingertip pulse oximeter to identify problem foods? What kind of readings do you see and when? How do the readings correlate with high or low blood sugar?"

    Sorry, I wasn't very clear. I use my pulse oximeter to track my pulse rate, which could also be done by a much cheaper blood pressure monitor. I use my pulse oximeter because I have it, and it is conveniently small.

    My pulse readings do correlate very well with my glucose meter readings for carbs, but  I don't really use it to directly track blood glucose, even though I have Type 2 diabetes. My most bothersome health symptoms are tachycardia and chest pain, and my pulse oximeter has helped me identify the problem foods, which are carbs and caffeine.

    As I said, my pulse readings correlate very well with my glucose meter responses for carbs, but my pulse oximeter also has the advantage of showing the effect of caffeine, which doesn't show up with my glucose meter.

    Also, my pulse oximeter shows my body's response almost immediately, whereas my glucose mete may take an hour or more.

    Thus, for me, with my concerns about tachycardia and chest pains, my pulse oximeter is much more useful than my glucose meter.

    Lest you think I got off topic, let me repeat that my pulse readings correlate closely with glucose readings, but much quicker.

    And again, a blood pressure monitor would give the same pulse information more cheaply.

    All this may apply only to me, or to diabetics who also have tachycardia. Everybody is different.

  • Anonymous

    12/26/2009 8:07:42 PM |

    Easily I assent to but I contemplate the brief should secure more info then it has.

  • Chloe

    12/27/2009 12:17:02 AM |

    Peter--Kip is on low carb, wet cat food once he was diagnosed in April (his 14th birthday!).  The vet wanted him on a prescribed mid-level carbohydrate food and an unbelievable amount of Vetsulin.  After finding www.felinediabetes.com, I started testing him, switched his food, and switched his insulin.  His BG levels run 40 to 120 (sometimes higher when he has a flare of pancreatitis) on 1U twice a day.  I have learned to keep shooting even when he runs normal sugars.  Congratulations to your friend for getting diet control.  I have been at this about 8 months with him and I am not sure he is going to go without insulin.  Still I am hopeful.  He has helped me on my diet though.  I can't cheat (really cheating myself) on mine anymore because I feel guilty about him AND since he often helps himself to my food I do not have anything he can't.  The household is strictly low carbohydrate: Meat, fat, and green vegetables. Glad to hear a good story about a cat becoming diet controlled. Human and feline we are all healthier these days.

  • Grandma S.

    12/27/2009 2:38:22 AM |

    Dr. Davis-How does the blood sugar rise correlate with taking Niacin and LDLs?  It raises my blood glucose levels, raises my HDL, and lowers my Trigl.  Thank you!

  • Anonymous

    12/27/2009 4:25:26 AM |

    Thanks Jim Purdy.  Just to clarify, exactly what are you seeing on your pulse oximeter when a food is "bad?"  For example, what is a good pulse reading and what is it when it is bad?  Do you track how long it takes to return to baseline?

    It is very interesting that it correlates with your glucose levels.  Are glucose levels high when your pulse is high and vice versa?  Even though the device is more expensive than a glucose monitor, you don't have the ongoing expense of the strips and of course don't have to stab yourself!  Of course a pulse oximeter wouldn't provide enough information for someone needing to treat diabetes but for those just curious about the effect of foods, it might suffice.

  • karl

    12/27/2009 4:42:40 AM |

    The question become is the small LDL the base risk factor or is it blood sugar (fructose?) .

  • Anonymous

    12/29/2009 3:53:23 PM |

    Good post and this post helped me alot in my college assignement. Thanks you for your information.

  • Anonymous

    1/8/2010 8:54:05 PM |

    I want not agree on it. I over precise post. Specially the appellation attracted me to be familiar with the unscathed story.

  • Anonymous

    1/17/2010 7:28:51 PM |

    I would also like further information about oats, I remember other posts in this blog that show oats as beneficial for reducing small LDL

  • Anonymous

    1/29/2010 7:33:32 AM |

    "Because blood sugar parallels small LDL, checking blood sugar can provide insight into how you respond to various foods and know whether glucose/small LDL have been triggered."

    I am very interested in pursuing this, but do you have a cite for this? I googled up small LDL particle size and didn't see anything about blood sugar being a good proxy for that, although I'm sure I missed seeing a lot.

    Also, I'm interested in more information on the effect of oats. I, too, have been under the impression that it is a healthy choice.

  • buy jeans

    11/3/2010 10:24:34 PM |

    However, you can easily and now cheaply check your blood sugar. Because blood sugar parallels small LDL, checking blood sugar can provide insight into how you respond to various foods and know whether glucose/small LDL have been triggered.

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